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Fig. 2 | BMC Geriatrics

Fig. 2

From: Prevalence and outcomes of rapidly progressive dementia: a retrospective cohort study in a neurologic unit in China

Fig. 2

Selected brain MRI images in participants with rapidly progressive dementia. The descriptive text of each image is shown with definitions of disease name, age, sex, disease course, and MRI data. (A) Creutzfeldt–Jakob disease: 72 years, female, 20 days. DWI image indicating the bilateral frontal and parietal cortex hyperintensities. (B) Neurosyphilis: 61 years, male, 2 months. T2WI demonstrating wide brain atrophy. (C) Tuberculous meningoencephalitis: 55 years, male, 1 month, T2WI demonstrating ventricular enlargement, paraventricular hyperintensities and increased vascular shadow. (D) Alzheimer’s disease: 75 years, male, 1 year, T2WI demonstrating wide brain atrophy and ventricular enlargement. (E & F) Neuronal intranuclear inclusion disease: 78 years, male, 2 months, T2WI demonstrating diffuse white matter hyperintensities and brain atrophy, and DWI images indicating hyperintensities involving corticomedullary junction in the frontal and parietal lobes. (G & H): CO poisoning: 61 years, female, 1 month, DWI and ADC images indicating diffuse sub-cortical hyperintensities. (I & J) Polyvinyl alcohol poisoning: 35 years, male, 1 month, T2WI images showing diffuse subcortical and globus pallidus hyperintensities. (K & L) Dichloroethane poisoning: 34 years, female, 10 days, T2WI showing diffuse subcortical and dentate nucleus hyperintensities. (M) Mitochondrial encephalomyopathy with lactic acidosis and stroke-like episode: 52 years, female, half a month. T2WI showing hyperintensity and local swelling on the bilateral temporal lobes. (N & O) Autoimmune encephalitis associated with LGI1 receptor: 47 years, male, 20 days, FLAIR showing hyperintensities on the bilateral hippocampus (N), and lesions disappeared after treatment at 6-month follow-up (O). (P & Q) Sjogren’s syndrome with central nervous system involvement: 45 years, male, 2 months, T2WI showing bilateral basal ganglia (caudate nucleus, globus pallidus and anterior thalamus) symmetrical hyperintensities (P), and enhanced sequence showing significantly enhanced lesions in patches (Q). (R) Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy: 62 years, male, 2 years, T2WI images showing hyperintensity on the pole of the bilateral temporal lobes

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